What Causes Neurotypicality?
What if difference never meant less?
When it comes to autism the question has been asked a million different ways: what causes it? Researchers chase after genes, toxins, parenting styles, diets and now even screen time. Whole careers (that's right, RFK Jr.) have been built on the search for âcausesâ even when no one can agree on what counts as proof.
But pause for a moment and flip the frame. Instead of asking what causes autism, let's ask this: what causes neurotypicality?
That question sounds silly, doesn't it? And silliness is the point. Because if you stop and sit with it, the absurdity reveals something important: autism only âneedsâ a cause because it is framed as a deviation. Neurotypicality is framed as normal so it never has to explain itself.
Letâs back up for a minute and explain some terms. Neurology is simply the way a personâs brain is wired â how it processes information, senses the world and communicates with others. Everyone has a neurology. Autism, ADHD and dyslexia are all forms of neurological difference. Neurotypicality is not the absence of neurology; it is the statistical majority profile, the cluster of developmental patterns that happen to be most common in the population.
Yet hereâs the twist: most neurotypical people have never even heard the word "neurotypical." They donât need to. Their neurology is the default, the vast majority, the yardstick against which everything else is measured. Thatâs how power works. Categories at the center donât need names because the world is already built around them.
Autistic people, by contrast, are always named, explained, diagnosed and pathologized. We are the ones who need an origin story, a justification for why we exist. Neurotypical people just get to exist without question.
But is neurotypicality really uncaused? Of course not. It is an outcome of human variation, just like autism. The majority of people inherit combinations of genes that shape their development toward what we call neurotypical pathways. And once those pathways become the majority, society reinforces them as the baseline. Schools, workplaces and families reward those traits and declare them natural.
Thatâs why neurotypicality feels invisible. Itâs not invisible because it has no cause. Itâs invisible because society declared it the standard. The ânormalâ child doesnât have to explain why they are normal. The autistic child does. The ânormalâ adult doesnât need to justify their way of communicating or relating. The autistic adult does. Most people don't even know we exist.
Shockingly, many people still imagine autism as something children âgrow out ofââbut autism is a lifelong neurotype. We are not broken; our brains are simply wired differently for the entirety of our lives.
When researchers insist on asking âwhat causes autism,â they are not really chasing scientific curiosity. They are upholding a system that assumes deviation needs explanation while the baseline goes unquestioned. Itâs the same logic that made left-handedness into a problem for centuries complete with causes, cures and corrections. Finally, society stopped pathologizing it. No one today writes grant proposals about ârisk factors for left-handedness.â We simply accept that some people, including me, are left-handed and account for about 10% of the population.
Imagine if we applied the same shift to differences in how the brain is wired overall. Instead of chasing causes, we would recognize autism as one expression of natural human brain variation. We would stop asking how to prevent it and start asking how to build a world that can make room for us.
Because in the end, autism doesnât need a cause any more than neurotypicality does. Both are simply outcomes of human variation. The only difference is that one has been crowned as the baseline and the other has been judged as a deviation, an abnormality.
So the next time you see a headline about âthe causes of autism,â flip the frame. Ask what causes neurotypicalityâand notice how quickly the logic collapses. That collapse is the sound of the frame breaking open.